Int J Stomatol ›› 2023, Vol. 50 ›› Issue (1): 19-24.doi: 10.7518/gjkq.2023020
• Periodontitis • Previous Articles Next Articles
Liu Tiqian(),Liang Xing(),Liu Weiqing,Li Xiaohong,Zhu Rui.
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1 | Frencken JE, Sharma P, Stenhouse L, et al. Global epidemiology of dental caries and severe periodontitis-a comprehensive review[J]. J Clin Periodontol, 2017, 44(): S94-S105. |
2 | Ma QY, Ma ZS, Liang MM, et al. The role of physical forces in osteoclastogenesis[J]. J Cell Physiol, 2019, 234(8): 12498-12507. |
3 | Li L, Han MX, Li S, et al. Cyclic tensile stress du-ring physiological occlusal force enhances osteoge-nic differentiation of human periodontal ligament cells via ERK1/2-Elk1 MAPK pathway[J]. DNA Cell Biol, 2013, 32(9): 488-497. |
4 | Zhu R, Zhang ZH, Lu BY, et al. Unloading of occlusal force aggravates alveolar bone loss in periodontitis[J]. J Periodontal Res, 2022, 57(5): 1070-1082. |
5 | Naert I, Duyck J, Vandamme K. Occlusal overload and bone/implant loss[J]. Clin Oral Implants Res, 2012, 23(): 95-107. |
6 | Fan JY, Caton JG. Occlusal trauma and excessive occlusal forces: narrative review, case definitions, and diagnostic considerations[J]. J Periodontol, 2018, 89(): S214-S222. |
7 | Hajishengallis G, Darveau RP, Curtis MA. The keystone-pathogen hypothesis[J]. Nat Rev Microbiol, 2012, 10(10): 717-725. |
8 | Hajishengallis G. Immunomicrobial pathogenesis of periodontitis: keystones, pathobionts, and host response[J]. Trends Immunol, 2014, 35(1): 3-11. |
9 | Usui M, Onizuka S, Sato T, et al. Mechanism of alveolar bone destruction in periodontitis-periodontal bacteria and inflammation[J]. Jpn Dent Sci Rev, 2021, 57: 201-208. |
10 | Martínez-García M, Hernández-Lemus E. Periodontal inflammation and systemic diseases: an overview[J]. Front Physiol, 2021, 12: 709438. |
11 | Passanezi E, Sant’Ana ACP. Role of occlusion in periodontal disease[J]. Periodontol 2000, 2019, 79(1): 129-150. |
12 | 祁海龙, 王斯璐. 慢性牙周炎伴咬合创伤患者龈沟液炎性因子表达及与骨代谢指标的相关性研究[J]. 现代检验医学杂志, 2021, 36(5): 164-168. |
Qi HL, Wang SL. Expression of inflammatory factors in gingival crevicular fluid and its correlation with bone metabolism in patients with chronic pe-riodontitis and occlusal Trauma [J]. J Mod Lab Med, 2021, 36(5): 164-168. | |
13 | Zhou SY, Mahmood H, Cao CF, et al. Teeth under high occlusal force may reflect occlusal trauma-associated periodontal conditions in subjects with untreated chronic periodontitis[J]. Chin J Dent Res, 2017, 20(1): 19-26. |
14 | Iwata M, Saito A, Kuroda Y, et al. Interdisciplinary therapy for severe periodontitis with Angle class Ⅱdivision 1 malocclusion: a case report with 7-year fo-llow-up[J]. J Am Dent Assoc, 2019, 150(11): 960-971. |
15 | Inchingolo AD, di Cosola M, Inchingolo AM, et al. Correlation between occlusal trauma and oral microbiota: a microbiological investigation[J]. J Biol Regul Homeost Agents, 2021, 35(2 ): 295-302. |
16 | Yoshinaga Y, Ukai T, Abe Y, et al. Expression of receptor activator of nuclear factor kappa B ligand relates to inflammatory bone resorption, with or without occlusal trauma, in rats[J]. J Periodontal Res, 2007, 42(5): 402-409. |
17 | Nakatsu S, Yoshinaga Y, Kuramoto A, et al. Occlusal trauma accelerates attachment loss at the onset of experimental periodontitis in rats[J]. J Periodontal Res, 2014, 49(3): 314-322. |
18 | Jia R, Yi YJ, Liu J, et al. Cyclic compression emerged dual effects on the osteogenic and osteoclastic status of LPS-induced inflammatory human periodontal ligament cells according to loading force[J]. BMC Oral Health, 2020, 20(1): 7. |
19 | El-Awady AR, Lapp CA, Gamal AY, et al. Human periodontal ligament fibroblast responses to compression in chronic periodontitis[J]. J Clin Periodontol, 2013, 40(7): 661-671. |
20 | Römer P, Köstler J, Koretsi V, et al. Endotoxins potentiate COX-2 and RANKL expression in compressed PDL cells[J]. Clin Oral Investig, 2013, 17(9): 2041-2048. |
21 | Li Y, Ling JQ, Jiang QZ. Inflammasomes in alveolar bone loss[J]. Front Immunol, 2021, 12: 691013. |
22 | DiDonato JA, Mercurio F, Karin M. NF-κB and the link between inflammation and cancer[J]. Immunol Rev, 2012, 246(1): 379-400. |
23 | Xu WZ, Lu Y, Yue JL, et al. Occlusal trauma inhi-bits osteoblast differentiation and bone formation through IKK-NF-κB signaling[J]. J Periodontol, 2020, 91(5): 683-692. |
24 | Lim WH, Liu B, Mah SJ, et al. Alveolar bone turnover and periodontal ligament width are controlled by Wnt[J]. J Periodontol, 2015, 86(2): 319-326. |
25 | Chang J, Sonoyama W, Wang Z, et al. Noncanonical Wnt-4 signaling enhances bone regeneration of me-senchymal stem cells in craniofacial defects through activation of p38 MAPK[J]. J Biol Chem, 2007, 282(42): 30938-30948. |
26 | Xu WZ, Lu Q, Qu MY, et al. Wnt4 regulates bone metabolism through IKK-NF-κB and ROCK signa-ling under occlusal traumatic periodontitis[J]. J Pe-riodontal Res, 2022, 57(3): 461-469. |
27 | Jia Q, Jiang WK, Ni LX. Down-regulated non-co-ding RNA (lncRNA-ANCR) promotes osteogenic differentiation of periodontal ligament stem cells[J]. Arch Oral Biol, 2015, 60(2): 234-241. |
28 | Lu Q, Xu WZ, Liu LY, et al. Traumatic compressive stress inhibits osteoblast differentiation through long chain non-coding RNA Dancr[J]. J Periodontol, 2020, 91(11): 1532-1540. |
29 | Zhu L, Xu PC. Downregulated LncRNA-ANCR promotes osteoblast differentiation by targeting EZH2 and regulating Runx2 expression[J]. Biochem Biophys Res Commun, 2013, 432(4): 612-617. |
30 | Grigoriadis AE, Wang ZQ, Cecchini MG, et al. C-Fos: a key regulator of osteoclast-macrophage li-neage determination and bone remodeling[J]. S-cience, 1994, 266(5184): 443-448. |
31 | Wang Y, Wang HY, Ye QS, et al. Co-regulation of LPS and tensile strain downregulating osteogenicity via c-fos expression[J]. Life Sci, 2013, 93(1): 38-43. |
32 | Belibasakis GN, Bostanci N. The RANKL-OPG system in clinical periodontology[J]. J Clin Periodontol, 2012, 39(3): 239-248. |
33 | Zhao B, Tumaneng K, Guan KL. The Hippo pathway in organ size control, tissue regeneration and stem cell self-renewal[J]. Nat Cell Biol, 2011, 13(8): 877-883. |
34 | Pan WY, Yang L, Li JL, et al. Traumatic occlusion aggravates bone loss during periodontitis and activates Hippo-YAP pathway[J]. J Clin Periodontol, 2019, 46(4): 438-447. |
35 | Wei W, Xue LL, Tan LY, et al. Inhibition of yes-associated protein dephosphorylation prevents aggravated periodontitis with occlusal trauma[J]. J Pe-riodontol, 2021, 92(7): 1036-1048. |
36 | Christgen S, Tweedell RE, Kanneganti TD. Programming inflammatory cell death for therapy[J]. Pharmacol Ther, 2022, 232: 108010. |
37 | Cheng R, Liu W, Zhang R, et al. Porphyromonas gingivalis-derived lipopolysaccharide combines hypoxia to induce caspase-1 activation in periodontitis[J]. Front Cell Infect Microbiol, 2017, 7: 474. |
38 | Jiang MY, Shang ZZ, Zhang T, et al. Study on the role of pyroptosis in bone resorption induced by occlusal trauma with or without periodontitis[J]. J Pe-riodontal Res, 2022, 57(3): 448-460. |
39 | Tang KM, Chen W, Tang ZH, et al. Role of the Hippo-YAP/NF-κB signaling pathway crosstalk in regulating biological behaviors of macrophages under titanium ion exposure[J]. J Appl Toxicol, 2021, 41(4): 561-571. |
40 | Park HW, Kim YC, Yu B, et al. Alternative wnt signaling activates YAP/TAZ[J]. Cell, 2015, 162(4): 780-794. |
41 | Xiong JH, Almeida M, O'Brien CA. The YAP/TAZ transcriptional co-activators have opposing effects at different stages of osteoblast differentiation[J]. Bone, 2018, 112: 1-9. |
42 | 廖安琪, 杨仁丽, 杨醒眉. 种植体周围炎的免疫应答机制及其影响因素的研究进展[J]. 口腔医学, 2021, 41(12): 1143-1147. |
Liao AQ, Yang RL, Yang XM. Research progress of the mechanism of immune response and influencing factors of peri-implantitis[J]. Stomatology, 2021, 41(12): 1143-1147. |